Abraham C, Cho JH (2009) Mechanisms of disease. N Engl J Med 361:2066–2078. https://doi.org/10.1056/NEJMra0804647
Lemberg DA, Day AS (2014) Crohn disease and ulcerative colitis in children: an update for 2014. J Paediatr Child Health 51(3):266–270. https://doi.org/10.1111/jpc.12685
Hammer T, Langholz E (2020) The epidemiology of inflammatory bowel disease: balance between East and West? A narrative review. Digest Med Res 3:48. https://doi.org/10.21037/dmr-20-149
NG, S. C., & CHAN, F. K. (2013). Infections and inflammatory bowel disease: challenges in Asia. Journal of Digestive Diseases, n/a. https://doi.org/10.1111/1751-2980.12091
Chouraki V, Savoye G, Dauchet L, Vernier-MassouilleG DJL, Merle V, Colombel JF (2011) e: a continuing increase in the 10-to 19-year-old age bracket (1988–2007). Aliment Pharmacol Therapeut 33(10):1133–1142. https://doi.org/10.1111/j.1365-2036.2011.04628.x
Khalyfa AA, Punatar S, Aslam R, Yarbrough A (2021) Exploring the inflammatory pathogenesis of colorectal cancer. Diseases 9(4):79. https://doi.org/10.3390/diseases9040079
Johnston DGW, Williams MA, Thaiss CA, Cabrera-Rubio R, Raverdeau M, McEntee C, Cotter PD, Elinav E, O’Neill LAJ, Corr SC (2018) Loss of microRNA-21 influences the gut microbiota, causing reduced susceptibility in a murine model of colitis. J Crohn’s Colitis 12(7):835–848. https://doi.org/10.1093/ecco-jcc/jjy038
Hu L, Huang Z, Wu Z, Ali A, Qian A (2018b) Mammalian plakins, giant cytolinkers: versatile biological functions and roles in cancer. Int J Mole Sci 19(4):974. https://doi.org/10.3390/ijms19040974
Hu, L., Xiao, Y., Xiong, Z., Zhao, F., Yin, C., Zhang, Y., …&Qian, A. (2017, September). MACF1, versatility in tissue-specific function and in human disease. In Seminars in cell & developmental biology (Vol. 69, pp. 3-8). Academic Press.doi: https://doi.org/10.1016/j.semcdb.2017.05.017.
Zhang YM, Wu QM, Chang LY, Liu JC (2020b) miR-34a and miR-125a-5p inhibit proliferation and metastasis but induce apoptosis in hepatocellular carcinoma cells via repressing the MACC1-mediated PI3K/AKT/mTOR pathway. Neoplasma 67(05):1042–1053. https://doi.org/10.4149/neo_2020_191019n1062
Won C, Kim BH, Yi EH, Choi KJ, Kim EK, Jeong JM et al (2015) Signal transducer and activator of transcription 3-mediated CD133 up-regulation contributes to promotion of hepatocellular carcinoma. Hepatology 62(4):1160–1173. https://doi.org/10.1002/hep.27968
Liu P, Tian W (2020) Identification of DNA methylation patterns and biomarkers for clear-cell renal cell carcinoma by multi-omics data analysis. PeerJ 8:e9654. https://doi.org/10.7717/peerj.9654
Afghani N, Mehta T, Wang J, Tang N, Skalli O, Quick QA (2017) Microtubule actin cross-linking factor 1, a novel target in glioblastoma. Int J Oncol 50(1):310–316. https://doi.org/10.1111/cas.13344
O’Brien J, Hayder H, Zayed Y, Peng C (2018) Overview of microRNA biogenesis, mechanisms of actions, and circulation. Front Endocrinol 9:402. https://doi.org/10.3389/fendo.2018.00402
Kim JS, Kim EJ, Lee S, Tan X, Liu X, Park S, Kang K, Yoon J-S, Ko YH, Kurie JM, Ahn YH (2019) MiR-34a and miR-34b/c have distinct effects on the suppression of lung adenocarcinomas. ExpMol Med. 51(1):9. https://doi.org/10.1038/s12276-018-0203-1
Zhao X, Li J, Ma J, Jiao C, Qiu X, Cui X, Wang D, Zhang H (2020) MiR-124a mediates the impairment of intestinal epithelial integrity by targeting aryl hydrocarbon receptor in Crohn’s disease. Inflammation 43(5):1862–1875. https://doi.org/10.1007/s10753-020-01259-0
Grimes C, Mohanan V (2015) Rescuing Nod2, an innate immune receptor of bacterial cell wall fragments, in Crohn Disease. FASEB J 29(S1). https://doi.org/10.1096/fasebj.29.1_supplement.571.19
Pierdomenico M, Cesi V, Cucchiara S, Vitali R, Prete E, Costanzo M et al (2016) NOD2 is regulated by Mir-320 in physiological conditions but this control is altered in inflamed tissues of patients with inflammatory bowel disease. Inflammatory Bowel Dis 22(2):315–326. https://doi.org/10.1111/febs.14482
Das MK, Andreassen R, And HTB, Furu K (2016) Identification of endogenous controls for use in miRNA quantification in human cancer cell lines. Genom Proteomics 13:63–68
Lin J, Redies C (2012) Histological evidence: housekeeping genes beta-actin and GAPDH are of limited value for normalization of gene expression. Dev Genes Evol 222(6):369–376. https://doi.org/10.1007/s00427-012-0420-x
Li WJ, Wang Y, Liu R, Kasinski AL, Shen H, Slack FJ, Tang DG (2021) MicroRNA-34a: potent tumor suppressor, cancer stem cell inhibitor, and potential anticancer therapeutic. Front Cell Dev Biol 9. https://doi.org/10.3389/fcell.2021.640587
Zhang L, Liao Y, Tang L (2019) MicroRNA-34 family: a potential tumor suppressor and therapeutic candidate in cancer. J Experiment Clin Cancer Res 38(1). https://doi.org/10.1186/s13046-019-1059-5
Boros É, Nagy I (2019) The role of microRNAs upon epithelial-to-mesenchymal transition in inflammatory bowel disease. Cells 8(11):1461. https://doi.org/10.3390/cells8111461
Voskuhl R (2011) Sex differences in autoimmune diseases. Biol Sex Differences 2(1):1. https://doi.org/10.1186/2042-6410-2-1
Ngo S, Steyn F, McCombe P (2014) Gender differences in autoimmune disease. Front Neuroendocrinol 35(3):347–369. https://doi.org/10.1016/j.yfrne.2014.04.004
El-Daly SM, Omara EA, Hussein J, Youness ER, El-Khayat Z (2019) Differential expression of miRNAs regulating NF-κB and STAT3 crosstalk during colitis-associated tumorigenesis. Mole Cell Probes 47:101442. https://doi.org/10.1016/j.mcp.2019.101442
Hou C, Wang D, Zhang L (2019) MicroRNA-34a-3p inhibits proliferation of rheumatoid arthritis fibroblast-like synoviocytes. Mole Med Rep 20(3):2563–2570. https://doi.org/10.3892/mmr.2019.10516
Ward CM, Li B, Pace BS (2016) Stable expression of miR-34a mediates fetal hemoglobin induction in K562 cells. Exp Biol Med 241(7):719–729. https://doi.org/10.1177/1535370216636725
Ma Y, Yue J, Zhang Y, Shi C, Odenwald M, Liang WG, Wu X (2017) ACF7 regulates inflammatory colitis and intestinal wound response by orchestrating tight junction dynamics. Nat Commun 8(1):1-16. https://doi.org/10.1038/ncomms15375
Rights and permissions
Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.
Disclaimer:
This article is autogenerated using RSS feeds and has not been created or edited by OA JF.
Click here for Source link (https://www.springeropen.com/)
